DocumentsDate added
Case report:-
Quadros Lydia S1*, Babu Arathy2, Bhat Nandini2, Ankolekar Vrinda Hari3
& D’souza Antony S4.
Affiliation:-
1*M.Sc.(Medical), 2Post graduates, 3MD Anatomy, 4MS Anatomy,Department of Anatomy, Kasturba Medical College, Manipal University, Madhavnagar, Manipal, Karnataka, India. – 576104.
Abstract:
A variation in the course and branches of the maxillary artery is well documented. In the present case, we came across variations in the branching pattern of the first and second parts maxillary artery in the right infratemporal fossa. It was noted that the Middle meningeal and accessory meningeal arteries took origin from the second part of maxillary artery and the deep temporal arteries aroused from the first part in common with the inferior alveolar artery and also from the second part of the maxillary artery. The second part of maxillary artery and its branches passed deep to the lateral pterygoid muscle and superficial to the branches of mandibular nerve. Rest of the branches had a normal origin. Such variations are of clinical importance for the surgeons in performing surgeries and also for the radiologists in interpretation of the radiological images.
Key Words: Infratemporal fossa; intra-arterial chemotherapy; maxillary artery.
References:
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Article citation:-
Quadros Lydia S et al. Anatomical variations in the branches of maxillary artery: A case report. Journal of pharmaceutical and biomedical sciences (J Pharm Biomed Sci.) 2013, July; 32(32): 1271-1273. Available at http://www.jpbms.info.
Copyright © 2013 Quadros Lydia S et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
Original article
1Shweta Kumari Singh, MDS student, 2Sumit Malhotra, MDS, ITS Dental College, Department of Periodontology and Oral Implantology, ITS Dental College, Muradnagar, Ghaziabad, India.
Abstract:
Background: Over the past decade, it has become more apparent that stress can negatively influence the oral health status, which can lead to increased amounts of dental plaque, gingival inflammation and more severe periodontitis.
Materials & method: Twenty students who were in fourth year B.D.S were recruited for the study. Test group (n=10) subjects were selected from final year students who were appearing for their examination. Control group (n=10) subjects were selected from non- examination going fourth year students. A clinical examination included assessment of Turesky-Gilmore-Glickman modification of Quigley-Hein Plaque index, Gingival Index, bleeding on probing, PPD and clinical attachment levels. Saliva and GCF were collected for each subject for analysis of salivary cortisol and GCF alkaline phosphatase (ALP) levels. Students registered their perceived stress on a visual analogue scale (VAS).
Results: Exam going students had higher amounts of dental plaque (P < 0.001), gingival inflammation (P < 0.001) and clinical attachment level (p < 0.001) compared with after the exams. The amount of ALP levels in GCF were significantly increased during the time of examinations. The median levels of cortisol in saliva was also significantly raised in exam going students compared to non-exam going students. The results from the VAS registration revealed a significant difference (P < 0.001) between the two groups.
Conclusion: Academic stress appears to affect periodontal health, shown by more plaque accumulation, gingival inflammation and increased amounts of ALP in GCF and cortisol in saliva.
Key Words: Academic stress; cortisol; dental plaque; alkaline phosphatase; periodontal diseases.
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Article citation:-
Singh Shweta Kumari & Malhotra Sumit. The effect of academic stress on gingival inflammation. Journal of pharmaceutical and biomedical sciences (J Pharm Biomed Sci.) 2013, July; 32(32): 1260-1264. Available at http://www.jpbms.info
Copyright © 2013 Singh Shweta Kumari & Malhotra Sumit. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited
Original article:-
Garima Dhariwal1 and Sumit Malhotra2*
Affiliations:-
1Garima Dhariwal, MDS, student, 2Sumit Malhotra MDS, Professor, ITS Dental College, Department of Periodontology and Oral Implantology, ITS Dental College, Muradnagar, Ghaziabad, India.
Abstract:-
Background: The main objective of the present study was to evaluate the efficacy of tetracycline fibre used along with scaling and root planing for the treatment of chronic periodontitis and to compare the results with those treated with scaling and root planing alone.
Method: A total of 20 patients (having at least one site with a pocket depth ≥ 5 mm) were selected for the study. Patients were divided randomly into Test group and Control group. In the control group, periodontal pockets were treated with scaling and root planing alone. In the test group periodontal pocket were treated with scaling and root planing followed by placement of tetracycline fibres. Clinical parameters, Gingival Index, Plaque Index (Turesky, Gilmore and Glickman modification of Quigley-Hein Plaque Index) and aspartate transaminase levels in gingival crevicular fluid were recorded at baseline, 15days and 30 days. Sulcus bleeding index, pocket probing depth and clinical attachment level were recorded at baseline and 30 days.
Results: Both treatment modalities were affective in improving clinical parameters over one month observation period. The combined antimicrobial and mechanical debridement therapy has shown better results as compared with scaling and root planing alone.
Conclusion: Application of tetracycline in modified collagen matrix following scaling and root planing might be beneficial in treatment of chronic periodontitis and improving periodontal parameters.
Key Words: tetracycline HCl fibres; aspartate transaminase; GCF.
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Article citation:-
Dhariwal Garima and Malhotra Sumit. Tetracycline fibers plus scaling and root planing Versus Scaling and root planing alone in chronic periodontitis: effect on GCF aspartate transaminase level. Journal of pharmaceutical and biomedical sciences (J Pharm Biomed Sci.) 2013, July; 32(32): 1265-1270. Available at http://www.jpbms.info.
Copyright © 2013 Dhariwal Garima and Malhotra Sumit. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
Original article:-
1Mohammed Umar Ansari, 1GangashankarGarg, 2L.R.Lodha, 3S. U. Qureshi & *4 S.Porwal
Affiliation:-
1Assistant Professor,2Professor and HOD,3Senior resident, Department of Anesthesiology, 4Associate professor, Department of surgery, SRG Hospital & Jhalawar Medical College Society, Jhalawar (Rajasthan),India.
Abstract:
Background: Propofol is a most frequently used intravenous (IV) anaesthetic today. It is used for induction, maintenance of anaesthesia and for sedation in and out-side operating room. Propofol provides rapid onset and offset with context sensitive decrement times approximately 10minutes when infused for less than three hours and less than 40 minutes when infused for 8 hours. At therapeutic doses, propofol reduces a moderate depressant effect on ventilation. It causes a dose- dependent decrease in blood pressure primarily through a decrease in cardiac output and systemic vascular resistance. A unique action of propofol is its antiemetic effect, which remain present at concentration less than producing sedation. The induction dose is 1-2 mg /kg for loss of consciousness with a maintenance infusion of 100-200 mcg/kg/min. For conscious sedation, rates of 25 to 75 mcg/kg/min are usually adequate.
Although pain on injection still remains a considerable concern for the anaesthesiologist. A number of techniques has been tried to minimize propofol-induced pain with variable results. Recently, a 5HT3 antagonist, ondansetron pre- treatment has been shown to reduce propofol- induced pain. The aim of our randomized, placebo-controlled, double blind study was to determine whether pre-treatment with intravenous ondansetron which is routinely used in our practice for prophylaxis of post – operative nausea and vomiting, would reduce propofol pain.
Methods: Eighty women, aged 18-50years, American society of anaesthesiologist grading (ASA) I-II, scheduled for various surgeries under general anaesthesia were randomly assigned to one of the two groups. One group received 2ml 0.9% sodium chloride while other group received 2ml ondansetron (2mg/ml) and was accompanied by manual venous occlusion for one min, then, 2ml propofol was injected through the same cannula. Patients were asked by a blinded investigator to score the pain on injection of propofol with a four point scale: 0= no pain, 1= mild pain, 2= moderate pain, 3= severe pain.
Results: Twenty four patients (60%) complaining of pain in the group pre- treated with normal saline as compared with six (15%) in the group pre-treated with ondansetron. Pain was reduced significantly in the ondansetron group.(P<0.05) severity of pain was also lesser in the ondansetron group compared with the placebo group(2.5%vs37.5%).
Conclusion: We conclude that pre-treatment with ondansetron along with venous occlusion for 1min for prevention of propofol induced pain was highly successful.
Key Words: Intravenous (I.V.) ondansetron; pain; pre- treatment; propofol injection.
Article citation:-
Ansari. M Umar, Garg. Gangashankar, Lodha L.R, Qureshi. S. U & Porwal. S. Pre- treatment with intravenous ondansetron to alleviate pain on propofol injection: A randomized, controlled & double-blind study. Journal of pharmaceutical and biomedical sciences (J Pharm Biomed Sci.) 2013, July; 32(32): 1274-1278.
Copyright © 2013 Ansari. M Umar, Garg. Gangashankar, Lodha L.R, Qureshi. S. U & Porwal. S. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
Original article:-
VermaMadhurima1, Gupta Ritu2, Porwal K Sanjay3*, Gupta Sanjeev4 & SwarnkarMadhusudan5
Affiliation:-
1Assistant Professor, 2Associate Professor, Department of Gynecology & Obstetrics, 3Associate Professor, Department of Surgery, 4Senior Resident Department of Anaesthesia, 5Assistant Professor, Department of PSM, Jhalawar Medical College, Jhalawar, Rajasthan, India.
Abstract:
Objective: To evaluate the prevalence of pelvic pathology among patient with uterine fibroid.
Material & method: The study was carried out in indoor patient of Zanana Hospital Jaipur Rajasthan a tertiary center attached to SMS medical collage & Hospital Jaipur, Rajasthan,India.
The study was carried out over a period of 18 month.Included 7348 cases of gynaecological admission out of which 508 cases were of fibroid uterus. All cases of fibroid of uterus were studied & managed surgically & were confirmed by histopathological examination. During surgery a systematic inspection of the pelvis was carried out to know the prevalence of associated pelvic pathology other then fibroid.
Results: In present series associated pelvic pathology were present in 189 cases (37.2%).Incidence of fibroid uterus come out to be 6.8% of gynaecological admission. The most common associated pelvic pathology were PID seen in 123 (24.2%) cases, next common were adenomyosis seen with 32 cases (6.2%),simple serous cyst 25 cases (4.9%) endometrosis 6 cases (1.1%) papillary adenoma with 2 cases (0.39%) dermoidcyst associated with one case ( 0.19%) of fibroid uterus.
Key Words: Uterine leiomyoma; fibroid; adenomyosis.
References:
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2. Evans P, Brunsell S. Uterine fibroid tumours: Diagnosis and treatment. American Family Physcian.2007;75:1503–1508.Pubmed
3. Lethaby A, Vollenhoven B. Fibroids (uterine myomatosis, leiomyomas) American Family Physcian.2005;71:1753–1756.Pubmed
Article citation:-
VermaMadhurima, Gupta Ritu, Porwal K Sanjay, Gupta Sanjeev & SwarnkarMadhusudan. To study the prevalence of pelvic pathology among patient with uterine myoma. Journal of pharmaceutical and biomedical sciences (J Pharm Biomed Sci.) 2013, July; 32(32): 1279-1281. Available at http://www.jpbms.info.
Copyright © 2013 VermaMadhurima, Gupta Ritu, Porwal K Sanjay, Gupta Sanjeev & SwarnkarMadhusudan. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.