DocumentsDate added
Research article:
Whyte Odigiyo Iyowuna., B. Sc., M. Sc1 , Kotingo Ebikabowei Lucky., MBBS, DMAS, FMAS,2,*,Kotingo Isuomo Victoria., B. Med Sc., MBBS.3
Affiliation:-
1Department of Science Foundation, School of Foundation Studies, Bayelsa State College of Health Technology, Otuogidi, Ogbia town, Bayelsa State, Nigeria
2Department of Obstetrics and Gynaecology, Federal Medical Centre, Yenagoa, Bayelsa State, Nigeria
3Department of Anatomy, Niger Delta University, Wilberforce Island, Amassoma, Bayelsa State, Nigeria
The name of the department(s) and institution(s) to which the work should be attributed:
1.Department of Science Foundation, School of Foundation Studies, Bayelsa State College of Health Technology, Otuogidi, Ogbia town, Bayelsa State, Nigeria
2.Department of Obstetrics and Gynaecology, Federal Medical Centre, Yenagoa, Bayelsa State, Nigeria
3.Department of Anatomy, Niger Delta University, Wilberforce Island, Amassoma, Bayelsa State, Nigeria
Address reprint requests to
Ebikabowei Lucky Kotingo.
Department of Obstetrics and Gynaecology, Federal Medical Centre, Yenagoa, Bayelsa State, Nigeria.
J Pharm Biomed Sci 2014; 04(06):509-514.
Article citation:
Whyte IO, Kotingo EL,Kotingo IV. Effects of Vitamin C supplementation on urinary Sodium and Potassium among exercising and non-exercising individuals in South West Nigeria. J Pharm Biomed Sci 2014; 04(05):509-508. Available at www.jpbms.info
ABSTRACT
Aim: The present study was carried out to investigate the effect of Vitamin C (300mg of Ascorbic acid) on the urinary excretion of sodium and potassium ion in exercising and non-exercising individuals.
Methodology: A total of 20 subjects all males were used for the study, 10 of whom were regularly exercising and the other 10 non-exercising (sedentary) individuals.
The Study lasted for a period of 6 weeks (42 days). The Subjects were all between the ages of 19-29. 20 to 26 (21.70 ± 1.26 years) for the exercising group, and 19 to 29 (23.90 ± 1.26 years) for the non-exercising group.
Results: The result obtained indicated that there was a significant reduction in urine potassium concentration by 6.31 ± 2.39 mmol/ L ( p ˂ 0.05) in non-exercising subjects. Exercising subjects like their non-exercising counterparts also showed a significant decrease in their urine potassium concentration by 6.62 ± 3.96 mmol/L ( p ˂ 0.05). In this case the significant decrease was accompanied by a significant increase in urine sodium concentration by 39.19 ± 11.65 mmol/ L (p ˂ 0.05).
Conclusion: The study indicates that Vitamin C supplementation facilitates the excretion of sodium while decreasing potassium expulsion via the kidney. This could possibly mean that Vitamin C can be classified as a potassium sparing diuretic. This action of Vitamin C could be used to explain its ability to decrease heart rate as well as blood pressure.
KEYWORDS: Vitamin C supplementation, Ascorbic acid, Urinary Sodium, Urinary Potassium, Exercising.
REFERENCES
1.Jacob, R.A., Vitamin C. In: Modern Nutrition in Health and Disease. Ninth Edition. Edited by Maurice Shils, James Olson, Moshe Shike, and A. Catharine Ross. Baltimore: Williams & Wilkins, 1999, p. 467-482.
2.Singh S,Edzard Ernst. Trick of Treatment: The Undeniable Facts about Alternative Medicine. WW Norton & Company. 2008; pp. 15–18. ISBN 978-0-393-06661-6.
3.SvirbelyJL, Szent-Györgyi A; Waugh. "The chemical nature of vitamin C". Biochem. J.1932; 26 (3): 865–70. Bibcode:1932Sci....75..357K. doi:10.1126/science.75.1944.357-a. PMC 1260981. PMID 16744896
4.Padayatty, S.J., Katz, A., Wang, Y., et al. Vitamin C as an antioxidant evaluation of it’s role in disease prevention. American college of Nutrition. 2003; 22 (1): 18 – 35 PMID 12569111.
5.Carl, F.S. The diuretics effect of ascorbic acid: preliminary report on its use in cardiac decompensation. JAMA. 1944; 124 (11): 700 – 701. Dol: 10.1001/ jama1944. 02850110024006.
6.Mohammed, A. A. The diuretic effect of vitamin C. Biochem J. 1937; 31 (2): 339 – 342. Retrieved from http:// www.ncbi. nLm. nih. gov/pmc/articles/PMC1266938/
7.Kaminski, M., and R. Boal. An effect of ascorbic acid on delayed-onset muscle soreness. Pain. 1992; 50:317-321.
8.Keith, R.E. Ascorbic Acid. In: Sports Nutrition Vitamins and Trace Minerals. Edited by Ira Wolinsky and Judy A. Driskell. New York: CRC Press, 1997, p. 29-45119-131.
9.Vasankari, T., U. Kujala, S. Sarna, and M. Ahotupa. Effects of ascorbic acid and carbohydrate ingestion on exercise induced oxidative stress. J. Sports. Med. Phys. Fitness.1998; 38(4): 281-285.
10.Howald, H., B. Segesser, and W.F. Korner. Ascorbic acid and athletic performance. Ann. N.Y. Acad. Sci. 1975; 258: 458-464.
11.Anne, W., Allison, G. (2001) Anatomy and Physiology in Health and Illness. 9th Edition. Edinburg. Elsevier Church Living Stone. 2001; p. 89-91. ISBN 0443064695.
12.Savini I, Rossi A, Pierro C. et al. "SVCT1 and SVCT2: key proteins for vitamin C uptake". Amino Acids. 2008;34 (3): 347–55. doi:10.1007/s00726-007-0555-7. PMID 17541511.
13.Rumsey SC, Kwon O, Xu GW. et al. Glucose transporter isoforms GLUT1 and GLUT3 transport dehydroascorbic acid. J. Biol. Chem.1997; 272 (30): 18982–9. doi:10.1074/jbc.272.30.18982. PMID 9228080.
14.May JM, Qu ZC, Neel DR, Li X. Recycling of vitamin C from its oxidized forms by human endothelial cells. Biochim. Biophys. Acta. 2003; 1640 (2–3): 153–61. doi:10.1016/S0167-4889(03)00043-0. PMID 12729925.
15.Packer L. "Vitamin C and redox cycling antioxidants". In Fuchs J, Packer L. Vitamin C in health and disease. New York: M. Dekker. 1997; ISBN 0-8247-9313-7.
16.Gerard, J.T., Sandra, R. G. Principles of Anatomy and Physiology, 8thEdition.Carlifornia.HarperCollins College Publishers. 1996; p. 896-897. ISBN 067399354X.
17.Klaus, D; Böhm, M; Halle, M. et al. "Die Beschränkung der Kochsalzaufnahme in der GesamtbevölkerungversprichtlangfristiggroßenNutzen" [Restriction of salt intake in the whole population promises great long-term benefits]. Deutsche MedizinischeWochenschrift (in German). 2009;3: S108–18. doi:10.1055/s-0029-1222573. PMID 19418415
18.Saito, T., Ishikawa, S., Higashiyama. et al. Inverse distribution of serum sodium and potassium in uncontrolled in patients with diabetes mellitus. Endocr J. 1999; 46(1): 75 – 80. PMID – 10426570.
19.Arthur. C.G., John, E. H. (2000). Text book of Medical Physiology. 10thEdition.Philadelphia, Pennsylvanna. W.B Saunders Company. 2000; p. 40-51. ISBN 072168677X
20.Rehrer, N.J. Fluid and Electrolyte balance in ultra-endurance sport. Med. 2001;31(10):701–15.
21.Breitwieser, G.E. Mechanism of potassium channel regulation. J Member. Biol.1996;152:1.
Source of support: None
Competing interest / Conflict of interest
The author(s) have no competing interests for financial support, publication of this research, patents and royalties through this collaborative research. All authors were equally involved in discussed research work. There is no financial conflict with the subject matter discussed in the manuscript.
Disclosure forms provided by the authors are available with the full text of this article at jpbms.info
Copyright © 2014 Whyte IO, Kotingo EL,Kotingo IV. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
Case report:
Muhittin Serkan Yilmaz1, Miray Baba1, Cemil Kavalcı2,*, Fevzi Yılmaz1, Müge Sonmez1,
Gulsüm Kavalci3,Bahattin Işik4, Bahadir Danişman5
Affiliation:-
1Numune Training and Research Hospital, Emergency Department, Ankara/Turkey
2Baskent University Faculty of Medicine, Emergency Department, Ankara/Turkey
3Yenimahalle state Hospital, Anesthesia Department, Ankara/Turkey
4Keciören Training and Research Hospital, Emergency Department, Ankara/Turkey
5Okmeydani Training and Research Hospital, Emergency Department, Istanbul/Turkey
The name of the department(s) and institution(s) to which the work should be attributed:
1.Numune Training and Research Hospital, Emergency Department, Ankara/Turkey
2.Baskent University Faculty of Medicine, Emergency Department, Ankara/Turkey
3.Yenimahalle state Hospital, Anesthesia Department, Ankara/Turkey
4.Keciören Training and Research Hospital, Emergency Department, Ankara/Turkey
5.Okmeydani Training and Research Hospital, Emergency Department, Istanbul/Turkey
*To whom it corresponds:-
Dr.Kavalci Cemil1, (Asoc.Prof)
Baskent University Faculty of Medicine, Emergency Department, Bahcelievler/Ankara/Turkey
Phone:+90 505 5762819
Fax;+90 312 223 6439
Abstract
Neuroleptic malignant syndrome (NMS) is a rare but potentially fatal complication of antipsychotic drug use. Its incidence varies between 0.07% and 2.2%. We aimed to report a case with NMS that developed after use of clozapine, an atypical antipsychotic agent.
Case: A 36-year-old man presented to emergency department with fever, sweating, difficulty in breathing, excessive muscle contractions, impaired speech, and altered consciousness. Approximately 10 days before he had been begun on clozapine (leponex) and ketiapin (seroquel) at a psychiatry clinic. Nearly 1 week after therapy onset he began to have loss of appetite, diffuse sweating, tremor in whole body including hands, and fever. He also began to have difficulty in breathing and altered consciousness three days before his presentation. On physical examination his body temperature was 42 0C, pulse rate 165 bpm, and blood pressure 90/50mmHg. CK-total: 973 U/L. Having reached a presumed diagnosis of neuroleptic malignant syndrome, we requested a neurology consultation.
Conclusion Risk of NMS should be taken into account at the time of starting antipsychotic medications and changing their doses in all patients.
Keywords: Emergency; neuroleptic malignant syndrome; atypical antipsychotic.
REFERENCES
1.Bottoni TN. Neuroleptic malignant syndrome: A brief review. Hosp Physician 2002; 38:58-63.
2.Kasantikul D, Kanchanatawan B. Neuroleptic malignant syndrome: a review and report of six cases. J Med Assoc Thai 2006; 89:2155-2160.
3.Kunz M, Gomes FA, Tramontina JF, Kapczinski F. Late-onset neuroleptic malignant syndrome in a patient using olanzapine. J Clin Psychopharmacol 2007;27:303-4.
4.Ananth J, Parameswaran S, Gunatilake S, Burgoyne K, Sidhom T. Neuroleptic malignant syndrome and atypical antipsychotic drugs. J Clin Psychiatry 2004;65:464-70.
5.American Psychiatric Association. Diagnostic and Statistical Manual of Mental Disorders, Fourth Edition Text Revision. Washington, DC, American Psychiatric Association, 2000.
6.Pope HG Jr, Cole JO, Choras PT, et al. Apparent neuroleptic malignant syndrome with clozapine and lithium. J Nerv Ment Dis 1986;174:493-5.
7.Mathews T, Aderibigbe YA. Proposed research diagnostic criteria for neuroleptic malignant syndrome. Int J Neuropsychopharmacol 1999;2:129-44.
8.Nierenberg D, Disch M, Manheimer E, et al. Facilitating prompt diagnosis and treatment of the neuroleptic malignant syndrome. Clin Pharmacol Ther 1991;50:580-6.
9.Rosebush PI, Stewart T, Mazurek MF. The treatment of neuroleptic malignant syndrome.Are dantrolene and bromocriptine useful adjuncts to supportive care? Br J Psychiatry 1991;159:709-12.
10.Miyaoka H, Shishikura K, Otsubo T, Muramatsu D, Kamijima K. Diazepamresponsive neuroleptic malignant syndrome: a diagnostic subtype? Am J Psychiatry 1997;154:882.
11.Seitz DP, Gill SS. Neuroleptic malignant syndrome complicating antipsychotic treatment of delirium or agitation in medical and surgical patients: Case reports and a review of literature. Psychosomatics 2009; 50:8-15.
12.Strawn JR, Keck PE, Caroff SN. Neuroleptic malignant syndrome. Am J Psychiatry 2007; 164:870-876.
Article citation:
Yilmaz MS, Baba M, Kavalci C, Yilmaz F, Sonmez M, Kavalci G, et al., A fatal neuroleptic malignant syndrome as a result of atypical Antipsychotic drug Use. J Pharm Biomed Sci 2014; 04(06):489-491.
Source of support: None
Competing interest / Conflict of interest
The author(s) have no competing interests for financial support, publication of this research, patents and royalties through this collaborative research. All authors were equally involved in discussed research work. There is no financial conflict with the subject matter discussed in the manuscript.
Copyright © 2014 Yilmaz MS, Baba M, Kavalci C, Yilmaz F, Sonmez M, Kavalci G, et al., This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
Research article:
Whyte Iyowuna Odigiyo., B. Sc., M. Sc1 , Kotingo Ebikabowei Lucky., MBBS, DMAS, FMAS,2,*,Kotingo Isuomo Victoria., B. MED Sc., MBBS.3
Affiliation:-
1Department of Science Foundation, School of Foundation Studies, Bayelsa State College of Health Technology, Otuogidi, Ogbia town, Bayelsa State, Nigeria.
2Department of Obstetrics and Gynaecology, Federal Medical Centre, Yenagoa, Bayelsa State, Nigeria.
3Department of Anatomy, Niger Delta University, Wilberforce Island, Amassoma, Bayelsa State, Nigeria.
The name of the department(s) and institution(s) to which the work should be attributed:
1.Department of Science Foundation, School of Foundation Studies, Bayelsa State College of Health Technology, Otuogidi, Ogbia town, Bayelsa State, Nigeria.
2.Department of Obstetrics and Gynaecology, Federal Medical Centre, Yenagoa, Bayelsa State, Nigeria.
3.Department of Anatomy, Niger Delta University, Wilberforce Island, Amassoma, Bayelsa State, Nigeria.
Address reprint requests to
Ebikabowei Lucky Kotingo.
Department of Obstetrics and Gynaecology, Federal Medical Centre, Yenagoa, Bayelsa State, Nigeria. med Sci 2014; 04(06):515-520.
Article citation:
Whyte IO, Kotingo EL,Kotingo IV. Effects of Vitamin C supplementation on blood oxygen saturation among exercising and non-exercising individuals in South West Nigeria. J Pharm Biomed Sci 2014; 04(05):515-520. Available at www.jpbms.info
ABSTRACT
Aim: The present study was carried out to investigate the effect of Vitamin C (300mg of Ascorbic acid) on blood oxygen saturation in exercising and non-exercising individuals.
Methodology: A total of 20 subjects all males were used for the study, 10 of whom were regularly exercising and the other 10 non-exercising (sedentary) individuals.
The Study lasted for a period of 6 weeks (42 days) during which each subject was given 3 tablets of Vitamin C daily. Each tablet contained 100mg of Ascorbic acid and 2mg of Aspartame. The Subjects were all between the ages of 19-29. 20 to 26 (21.70 ± 1.26 years) for the exercising group, and 19 to 29 (23.90 ± 1.26 years) for the non-exercising group.
Results: The result obtained showed that there was no significant difference in the blood oxygen saturation after Vitamin C supplementation. However a significant change was observed when the changes in blood oxygen saturations for exercising and non-exercising where compared. (Non-exercising SPO2=1.86 ± 1.15, Exercising SPO2= 0.39 ± 0.14, p < 0.05).
Conclusion: In this study series, Vitamin C supplementation showed no effect on a blood oxygen saturation level in both exercising and non-exercising individuals. However, a significant association was observed when the change in the exercising subjects was compared with the non-exercising subjects. Vitamin C has been widely investigated as it relates to several biological factors yet with scanty literature on its relationship with oxygen saturation.
KEYWORDS: Vitamin C supplementation, Ascorbic acid, Blood oxygen saturation, Exercising subjects, Non-exercising subjects.
REFERENCES
1.Groff, J.E., Gropper, S.S., and Hunt, S.M. The Water Soluble Vitamins in Advanced Nutrition and Metabolism. Minneapolis: West Publishing Company. 1995; p222 – 237.
2.Howald, H., Segesser, B., and Korner, W.F. Ascorbic acid and athletic performance. Annuals of the New York Academy of Sciences.2006; 258, 458 – 464. Retrieved from http://onlinelibrary. Wiley.com. Doi: 10.1111/j.1749 – 6632. 1975 tb29304.x. Retrieved from http: // Americancollegeofnutrition.org/content/full/22/1/18.
3.Padayatty, S.J., Katz, A., Wang, Y., Eck, P., Kwon, O., Lee, J.H., Chen, S, Corpe, C., Dutta, A., Dutta, S.K., Levine, M. Vitamin C as an antioxidant evaluation of it’s role in disease prevention. American college of Nutrition. 2003; 22 (1): 18-35 PMID 12569111.
4.Jacob, R.A., Vitamin C. In: Modern Nutrition in Health and Disease. Ninth Edition. Edited by Maurice Shils, James Olson, Moshe Shike, and A. Catharine Ross. Baltimore: Williams & Wilkins, 1999, p. 467-482.
5.Lind J. A Treatise of the Scurvy. London: A. Millar.http://www.bruzelius. info/ Nautica/ Medicine/Lind. Html. 1753.
6.Carpenter KJ. The History of Scurvy and Vitamin C. Cambridge, UK: Cambridge University Press Book reviews Nature (1986);324:177 J Hist Med Allied Sci (1987);42:386-7 Med Hist (1987);31:231-2 Yale J Biol Med. 1989;62(3):333–334 CMAJ (1986)135(10):1160
7.Hallberg L, Brune M, Rossander L. The role of vitamin C in iron absorption. Int J Vitam Nutr Res Suppl; 1989; 30: 103 – 8. PMID: 2507689.
8.Elaine, N.M., Katja, H. Human Anatomy and Physiology. 8th Edition. San Francisco, Pearson Benjamin Cummings (Pearson Education Inc). ISBN-13: 978-0-321-60261-9. ISBN-10: 0-321-60261-7. 2010; p. 638-642
9.Hypoxemia (Low blood Oxygen). Mayo Clinic. Retrieved October 28, 2013 from http://www.mayoclinic.org/symptoms/hypoxemia/basic/defination/sym - 20050930
10.Amperordirect.http://www.amperodirect.com/pc/help-pulse-oximeter/z-what is oxygen saturation.html. 2011.
11.O’Driscoll, B.R, Howard, L.S., Davison, A.G. BTS guideline for emergency oxygen use in adult patients. British Thoracic Society. 2008
12.Odunuga, A.C., Jaja, S.I, Ojo, G.O. Some Ventilatory Parameters in well trained Nigerian Atheletes. Nigerian Journal of Physiological Sciences. 1989; 5:7-16.
13.Amy, D. Oxygen Saturation Exercise. Retrieved October 30, 2013 from http; // www. Live strong. Com / article / 278796 – Oxygen Saturation exercise.
14.Fayed, N.M., Modrego, P.J., Morales, H. Evidence of brain damage after high – altitude climbing by means of magnetic resonance imaging. The American Journal of Medicine (Elsevier). 2006; 199 (2): 168.
15.Keith, R.E. Ascorbic Acid. In: Sports Nutrition Vitamins and Trace Minerals. Edited by Ira Wolinsky and Judy A. Driskell. New York: CRC Press. 1997; p. 29-45119-131.
Copyright © 2014 Whyte IO, Kotingo EL, Kotingo IV. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
Source of support: None
Competing interest / Conflict of interest
The author(s) have no competing interests for financial support, publication of this research, patents and royalties through this collaborative research. All authors were equally involved in discussed research work. There is no financial conflict with the subject matter discussed in the manuscript.
Disclosure forms provided by the authors are available with the full text of this article at jpbms.info.
Research article:
Elgylani,Hind, M.1,*& Wafa, Elhag, I,PHD2
Affiliation:-
1Msc-Student, faculty of Medical Laboratory Sciences- ALNeelain University,Khartoum, Sudan
2Assistant Professor, Microbiology department, faculty of Medical Laboratory Sciences-ALNeelain University, Khartoum ,Sudan
The name of the department(s) and institution(s) to which the work should be attributed:
Faculty of Medical Laboratory Sciences-ALNeelain University, Khartoum, Sudan
Microbiology department, faculty of Medical Laboratory Sciences-ALNeelain University ,Khartoum,Sudan
Address reprint requests to
Hind Mohamed Elgylani.
Microbiology Department, Faculty of Medical Laboratory Sciences AL-Neelain University, Kharoum, Sudan or at drhano4@yahoo.com
J Pharm Biomed Sci 2014; 04(06):559-562.
Article citation:
Elgylani, Hind M.& Wafa, Elhag I. Frequency of Aspergillus Spp. among chronic rhino sinusitis patients attending Khartoum ENT hospital,Sudan. J Pharm Biomed Sci 2014;04(05):559-562. Available at www.jpbms.info
ABSTRACT
Back ground: Chronic rhino sinusitis (CRS) is a common disease with significant morbidity and health care cost, although the medical and surgical treatments for CRS have improved markedly over the past few decades.
Objective: This study aimed to determine the frequency of Aspergillosis in the paranasal cavities in patients who had chronic rhino sinusitis.
Methodology: The Specimen was collected and processed by direct microscopy (using 10%KOH), conventional culture technique, observation of growth (colony configuration), lacto phenol cotton blue staining, and histopathological staining technique.
Results: Out of the total 50 nasal polyp specimens 13(26%), were positive for Aspergillosis by direct microscopy and cultural technique, also histopathological technique confirmed the positivity.
Among the total 13(26%) isolated fungi, (46.2%) were Aspergillus Niger, (38.5%) were Aspergillus flavus, and (15.3%) were Aspergillus fumigatus.
The remaining 37(74%) were negative with previously mentioned techniques.
Conclusion: Aspergillus spp. responsible for (26%) of chronic rhino sinusitis and most of them were due to Aspergillus Niger.
KEYWORDS: Chronic Rhino sinusitis; Mycotic chronic rhinosinusitis.
REFERENCES
1.Polzehl D, Moeller P, Riechelmann H, Perner S. Distinct features of chronic rhino sinusitis with and without nasal polyps. Allergy. 2006 ; 61:1275 - 1279.
2.Settipane GA. Epidemiology of nasal polyps. Allergy Asthma Proc 1996 ; 17: 231-236.
3.Demoly P, Crampette L, Daures JP. National survey on the management of rhinopathies in asthma patients by French pulmonologists in everyday practice. Allergy. 2003;58:233–238.
4.Kuhn F, Javer A. Allergic fungal rhino sinusitis: perioperative management, prevention of recurrence, and role of steroids and antifungal agents. Clin Otol North Am 2000 ; 33:419-432.
5.Anand VK. Epidemiology and economic impact of rhino sinusitis. Ann Otol Rhino Laryngol Suppl. 2004 ; 193 : 3–5 .
6.Stankiewicz JA, Chow JM. Nasal endoscopy and the definition and diagnosis of chronic rhino sinusitis. Otolaryngology Head Neck Surg. 2002 ; 126 : 623 - 627 .
7.Iro H, Mayr S, Wällisch C, Schick B, Wigand ME. Endoscopic sinus surgery :its subjective medium-term outcome in chronic rhino sinusitis. Rhinology. 2004 ; 42 : 200-206.
8.Stammberger H, Posawetz G. Functional endoscopic sinus surgery concept, indications and result of the MesserKlingers technique Eur Arch Otorhinolangol 1990;247:63-76.
9.Becker DG. Sinusitis. J Long Term Eff Med Implants 2003 ; 13: 94-175.
10.Manning SC, Mabry RL, Schaefer SD, Close LG. Evidence of IgE -mediated hypersensitivity in allergic fungal sinusitis. Laryngoscope. 1993 ; 103:717-721.
11.Stammberger H, JaKse R, Beaufort F. Aspergillosis of the paranasal sinuses .X- ray diagnosis histopathology and clinical aspect. Ann Otol Rhino Laryngol 1984;93:251-256.
12.Min YG, Kim HS, Kang MK, Han MH.Aspergillus sinusitis:clinical aspect and treatment outcomes.Otol Head Neck Surgery 1996;115:49-52.
13.Casas G. Contribucion al estudio de Aspergillus y aspergillosis .Kasmera 1972;4:94-107.
14.Kinsella JB, Bradfield JJ, Gourley WK, Calhoun KH,RasseKh CH .Allergic fungal sinusitis.Clin Oto North Am. 1996 ; 21: 389-392.
15.Waxman JE,Spector JG,Sale SR,katzenstein AL.Allergic Aspergillus sinusitis concept in diagnosis and treatment of new clinical entity .Laryngoscope 1987 ; 9:260-261.
16.Magaldi S, Camero. T. Aspergillosis broncopulmonar alergica en Venezuela.Arch.Med Troo1998 ; 2:16- 21.
17.Ponikau J, Sherris D, Kern E et al. The diagnosis and incidence of allergic fungal sinusitis Mayo Clin Proc 1999;74:877-884.
18.Chakrabarti A, Sharma SC, Chander J. Epidemiology and pathogenesis of paranasal sinus mycoses .Otol Head Neck Surg1992 ; 107: 745 -750.
19.Corey JP, Romberger CF, Shaw GY. Fungal diseases of the sinuses.Otol Head Neck Surg 1990;103:1012-5.
Copyright © 2014 Elgylani, Hind M. & Wafa, Elhag I. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
Source of support: None
Competing interest / Conflict of interest
The author(s) have no competing interests for financial support, publication of this research, patents and royalties through this collaborative research. All authors were equally involved in discussed research work. There is no financial conflict with the subject matter discussed in the manuscript.
Disclosure forms provided by the authors are available with the full text of this article at jpbms.info
Case report
Ankit Srivastava.,MDS1,Puneet2, Chetna Arora.,MDS3,*,Preetinder Singh4 ,Radhika Lekhi5
Affiliation:-
1 Senior Lecturer, Department of Pedodontics and Preventive Dentistry
SGT Dental College, Hospital and Research Institute, Village Bhudera, Gurgaon,India
2Post-graduate Student, Department of Conservative Dentistry and Endodontics, Government Dental College and Hospital, Patiala,India
3Senior lecturer, Department of Conservative Dentistry, Swami Devi Dyal Dental College, Barwala, Panchkula, Haryana,India
4Reader, Department of Periodontics and Implantology, Swami Devi Dyal Dental College, Barwala, Panchkula, Haryana,India
5Senior lecturer, Department of Conservative Dentistry and Endodontics, Sudha Rastogi College of Dental Sciences and Research, Faridabad,India
The name of the department(s) and institution(s) to which the work should be attributed:
Department of Conservative Dentistry and Endodontics, Government Dental College and Hospital, Patiala, India
Address reprint requests to
Dr. Chetna Arora.
Senior lecturer, Department of Conservative Dentistry
Swami Devi Dyal Dental College, Barwala, Panchkula, Haryana,India
J Pharm Biomed Sci 2014; 04(06):521-524.
Article citation:
Srivastava A,Puneet,Chetna A,Singh P,Lekhi R. Endodontic Retreatment of maxillary canine with two separate root canals: A case report. J Pharm Biomed Sci 2014; 04(05):521-524.Available at www.jpbms.info
ABSTRACT
Failure to identify and adequately fill a canal may be a major reason in the failure of root canal herapy. A clear understanding of all the potential aberrant canal morphologies is essential to ensure predictable success in completing complex cases. This case report describes re-treatment of a maxillary canine with two canals.
KEYWORDS: Endodontic retreatment; Upper canine; Anatomical variations; Vertucci’s Type II.
REFERENCES
1.Krasner P, Rankow H J Anatomy of the Pulp-Chamber Floor. J Endod 2004; 30: 5-16.
2.Vertucci F.J. Root canal morphology and its relationship to endodontic procedures. Endod Topics 2005; 10: 3–29.
3.Hargreaves KM, Cohen S. Pathways of the Pulp, 10th edition, Mosby, 2011; 10th edition.
4.Giuseppe C, Elio B, Arnaldo C. Missed anatomy: frequency and clinical impact. Endod Topics 2009; 15: 3-31.
5.Gandhi B, Majety KK, Gowdra. Root Canal treatment of bilateral three-rooted maxillary first premolars. J Orofac Sci 2012; 4 (1): 56-59.
6.Vertucci FJ. Root canal anatomy of the human permanent teeth. Oral Surg, Oral Med, Oral Pathol, Oral Radiol and Endod1984; 58: 589 -99.
7.Weisman MI. A rare occurrence: a bi-rooted upper canine. Aus Endod J 2000; 26: 119-20.
8.Onay OE, Ungor M. Maxillary Canines with two root canals. Hacettepe Dis Hekimligi Fakultesi Dergisi. 2008; 32: 20–24.
9.Shin DR, Kim JM, Kim DS, Kim SY, Abbott PV, Park SH. A maxillary canine with two separated root canals: A case report. J Korean Acad Cons Dent 2011 Sep; 36(5): 431-35.
10.Alapati S, Zaatar EI, Shyama M, Al-Zuhair N. Maxillary canine with two root canals. Med Princ Pract 2006; 15: 74-76.
Copyright © 2014 Srivastava A, Puneet, Chetna A, Singh P, Lekhi R. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
Source of support: None
Competing interest / Conflict of interest
The author(s) have no competing interests for financial support, publication of this research, patents and royalties through this collaborative research. All authors were equally involved in discussed research work. There is no financial conflict with the subject matter discussed in the manuscript.
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